Ok I will start to explain some aspects of the Level 2 test using notes from my course and text from FaCT-Canada...
Recovery Intensity (RI ) upper limit is established by:-
Polar Ownzone test , but you take only the lower number, which is the HR where the HRV (heart rate variability) is equal to 4 ms. 2 possibilities : a model with ownzone on it or a model with actual displaying RLX factor in msec.
With Paul I started to monitor his RLX @ rest as you can see it's high at rest on the bike @ 22.8ms for HR 61 and drops to 4ms @ HR 101. Above 4ms there's no stress to the cardiovascular system.
The Recovery Intensity @ 4ms varies day to day with training and you can see here how mine changes due to training stimmulus (see ownzone value on spreadsheet) :-
http://www.fact-canada.com/discus/messa ... 1209156329Now this limit is the start of the next zone which is the Basic Intensity STF zone with mainly STF fibers involvement.
We are absolutely aware , that there are no clear lines in between the different metabolic reactions , but a steady smooth in each other moving of the metabolic demands. Nevertheless , there are certain preferences from the body on how to produce the best metabolic approach in a current energy demanding situation..
The Basic Intensity STF zone would be the metabolic situation , where FFA is the dominant energy supplier together with O2 ( Taking place in the mitochondria ( mitochondrial respiration ).
The FFA metabolic pathway is using somewhat more oxygen than the glycolytic pathway (glycolysis is the sequence of reactions that converts glucose into pyruvate with the concomitant production of a relatively small amount of adenosine triphosphate (ATP)) even if the flux is still in a steady state mode , so lactate production is still in a range , where lactate as well as H+ are "controlled in the working muscle cell.
More oxygen means , if you increase the intensity like in a step test you may produce over time a situation , where you may see a slightly but measurable drop of O2 Sat.
Now in athletes with a clear difference in FFA and glycolytic activity (STF verusu FTF ) we see first a slightly drop in O2 Sat , followed by an increase in breathing frequency. This increase may be due to a lower O2 Sat and therefor a slightly higher PaCO2 (partial pressure of carbon dioxide in the arterial blood), which is a potent stimulator of respiratory rate.
Now if the additional breathing frequency is good enough to keep enough O2 coming into the working muscle , as well the respiration ( expiration ) is strong enough to release the CO2 (controlling for the moment the H+ production , we see still not an increase in lactate in the system ( because not yet "overflow" of H+ in the cytosol and therefor no need yet of lactate transporter to move as well H+ into the system.. Once we see a trend of lactate increasing in the system , we know , that the "steady state of H+ production and removal in the muscle cell is over and we can use the lactate as an indirect marker of acidic situation in the muscle. Later you will see how out of this situation we produced the name LBP for lactate balance point, which is the upper limit of the whole AZ zone . ( and AZ zone for aerobic zone and this zone is again as we started out split into BI-STF and BI-FTF zone. )
We dont always can see a very clean cut between BI-STF and BI-FTF , but in the most cases you still have a very good idea.
So BI-STF zone limits : RI upper limit and the intensity or HR where you see first a drop of O2 and in the next step a climb in breathing frequency .
Below now a typical protocol:
Watt/Breathing frequency/ O2 Sat/HR
100/ 12 / 98 / 128
120/ 12 / 97 / 135
140/ 12 / 98 / 141
160/ 12 / 96 / 149
180/ 16 / 96 / 156
200/ 16 /94 / 163
So the drop O2 is by 149 and the reaction by 156 of the bpm.
BI-STF zone upper limit is 149.
We tried to make practical confirmation tests , and if this athlet keeps his HR below 145 we can train every day very long ( 3 - 5 hours , and never see in the FaCT test an incline in lactate after breakfast ( carbo loading or very minimal. ) in comparison to training above 149 but below LBP where we see no lactate production or increase during the training , but an increase of lactate the next morning after breakfast, as well as a drop in blood glucose . which would indicate an involvement of Glucose in the energy production , but still in a "steady state of H+ production - still controlled in the working muscles. )
Rob