Resting lactate so high ?
2.1 as i can remeber is actually not that high and not that uncommon.
You can't compare it with teh actual number at the LBP , as the number on the LBP is not the actual lactate you would have just by that intensity as it depends on how high it was at the end of the test and the time between each sample taking:
Example
Lactate at end of the test 7.6
Now you drop to 5.4
3.4 and go up to 3.9
You may have the same lactate at the end 7.6
but now you take the lactate 1 - 2 min later ( so longer steps ) but by the same HR
Now you drop perhaps to 3.6
1.9 and go up to 2.7 So first LBP lacte by 3.4
and the second example by 1.9.
Non of them is abn absolute value but a picture of the lactate trend at the specific moment.
The 2.1 pre test lactate can be caused by many different reasons.
here 3 most common explanations.
1. ( no critic on Rob ) but it could have been a measurment mistake.
But with 2.1 unlikely . If I am not sure why I take a second sample and charge the cleint 4 x the fee (
2. Any intake of glucose just short bnefore the tes 10 min =_ will often increase the resting lactate.
3.
If a person was sick and the glucose stores where very empty liver and muscles than we see often higher lactate values as a sign of lactate shuttle function.
4.
If somebody is a bit nerveous ( exited ) he has a tendency to release adrenalin ) which will release as a reaction more glucose from the liver glycogen storage and the higher blood sugar lvel now with liitle activity yet can trigger again a blood shuttle reaction , where the lactate will work as the "truck" to shuttle energy ( in this case glucose in different body areas like liver and or other body parts.
Gladden and Brooks show nice evidence, that laxctate is one possiblity to use stored glucos in the muscles ( muscle glycogen ) in other parts of teh body.
Traditionaly we learned , that once you stored glucoe in the muscle, that's it , it only can be used there and not anymore in other areas like for the brain or arm glucogen can't be used in the legs.
As so often we seem to where wrong . Thanks to lacatate we may be able to shuttle leg glycogen to the arms and visa versa. How by converting glucose in the legs into lactate and now the body can move it to other areas.
It is one of Noakes puzzle answers, of survival of the body.
The body seems not to give up if there is any other way to keep itself alive. Brooks lactate shuttle theory works nicely into the CGM model and answers many different open questions.